— Tobias B. Huber, MD
Created with BioRender
We aim at a better understanding of podocytopathies, the identification of complex principles of kidney diseases, immune -tissue interactions and interorgan signaling. For this purpose, our lab is advancing the following research areas:
With this, our aim is to identify new approaches for the prevention, halt and cure of kidney diseases.
Chair of the Center of Internal Medicine
Director of the III. Department of Medicine
1992 - 1999
School of Medicine (10/1992-05/1999), Universities of Freiburg, Vienna (Austria) and South Florida (Tampa, USA)
2020 - 2020
Chair of the Center of Internal Medicine, UKE
Chair of the III. Department of Medicine, UKE
Chief of the Division of Chronic Kidney Disease, University Medical Center Freiburg (UKF), Germany
W3 Heisenberg Professor, UKF
German Board Certification for Internal Medicine (2008) and Nephrology (2011)
USMLE US-American Full Medical Licence
M.D. in Physiology (summa cum laude), Department of Physiology, UKF
2015 - 2017
Deputy Chair, Department of Medicine IV, UKF
2011 - 2015
Senior Attending (Geschäftsführender Oberarzt), UKF
2008 - 2011
Attending Physician, Department of Medicine IV, UKF
2001 - 2003
Fellow in Medicine, Department of Medicine IV, UKF
1999 - 2001
Residency, Department of Medicine IV, UKF
2013 - 2017
W3 Heisenberg Professorship, UKF
2006 - 2011
Emmy Noether Programme – Phase II / Group leader, UKF
2003 - 2006
Emmy Noether Programme – Phase I / Postdoctoral Fellow, Immunology & Pathology, Washington University St. Louis, USA (Laboratory of Prof. Dr. A. Shaw)
2021 - 2020
Elected Member of the National Academy of Sciences – Leopoldina-
Elected Member of the American Association of Physicians
Chair Else Kröner-Fresenius Clinician-Scientist Kolleg “iPRIME-CS”, UKE
Co-Chair BMBF-Advanced Clinician Scientist Program “iSTAR”, UKE
Speaker (together with U. Panzer) of the CRC 1192
DFG-Senate Commission on Key Questions in Clinical Research
Chair Else Kröner-Fresenius MD-Thesis Kolleg “iPRIME”, UKE
Co-Chair Else Kröner-Fresenius Kolleg “NAKSYS”, UKF
Recipient of the European Research Council Grant
Elected Member of the American Society of Clinical Investigation
Young Investigator Award of the American Society of Nephrology (Considered the worldwide highest award for renal research < 45 years)
Franz-Volhard Award of the German Renal Society
Hans U. Zollinger Research Award of German Renal Society
Associated Member of the BIOSS Excellence Cluster, Freiburg
Principal Investigator of the SGBM Graduate School, Freiburg
Carl Ludwig Award of the German Renal Society
Molecular consequences of SARS-CoV-2 liver tropism.
Pro-cachectic factors link experimental and human chronic kidney disease to skeletal muscle wasting programs.
Podocytes maintain high basal levels of autophagy independent of mtor signaling.
Decoding myofibroblast origins in human kidney fibrosis.
SARS-CoV-2 renal tropism associates with acute kidney injury.
Multiorgan and Renal Tropism of SARS-CoV-2.
Anaerobic Glycolysis Maintains the Glomerular Filtration Barrier Independent of Mitochondrial Metabolism and Dynamics.
ARP3 Controls the Podocyte Architecture at the Kidney Filtration Barrier.
The FERM protein EPB41L5 regulates actomyosin contractility and focal adhesion formation to maintain the kidney filtration barrier.
mTORC2 critically regulates renal potassium handling.